CorrespondenceValidation of schizophrenia gene expression profile in a preclinical model of maternal infection during pregnancy
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Conflict of interest
The authors declare no conflict of interest.
Acknowledgements
This study was funded by NIH R01 MH084894 and R01 MH111940 grants and SAF2010-20840-C02-01/02 and SAF2013-46292-R from MINECO (Spain).
References (10)
- et al.
Decoding the signaling of a GPCR heteromeric complex reveals a unifying mechanism of action of antipsychotic drugs
Cell
(2011) - et al.
A unifying hypothesis of schizophrenia: abnormal immune system development may help explain roles of prenatal hazards, post-pubertal onset, stress, genes, climate, infections, and brain dysfunction
Med. Hypotheses
(2010) - et al.
Schizophrenia gene expression profile reverted to normal levels by antipsychotics
Int. J. Neuropsychopharmacol.
(2014) - et al.
Transcriptome fingerprints distinguish hallucinogenic and nonhallucinogenic 5-hydroxytryptamine 2A receptor agonist effects in mouse somatosensory cortex
J. Neurosci.
(2003) - et al.
Identification of a serotonin/glutamate receptor complex implicated in psychosis
Nature
(2008)
There are more references available in the full text version of this article.
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These authors supervised equally this work.
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